Meeting report from the Euroscicon event: 3rd October 2013, Cineworld: The O2, London, UK
Meeting Chair: Dr. Gary Warnes
Blizard Institute, Barts & The London School of Medicine & Dentistry, Queen Mary University
Author: Dr Eleftherios Karanasios, Babraham Institute,
Babraham Research Campus, Cambridge
Editors: Dr Astrid Englezou and Dr Shara Cohen
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This one-day meeting “Analysis of Autophagy Regulation: Discussion of recent research and new technologies” organized by Euroscicon, was held on the 3rd of October 2013, in London, UK. Autophagy is a membrane trafficking pathway that cells activate under diverse stress conditions to deliver cytosolic material for degradation to lysosomes. Different techniques were presented, to study the regulation of autophagy, with special emphasis on imaging and flow cytometric assays. Speakers from diverse disciplines presented data, which included the basic mechanisms of autophagosome formation and the role of autophagy in human physiology and disease, including neurodegeneration, cancer and infectious diseases. The understanding of autophagy and its implications for human health and disease has come a long way in the last few years. Novel technologies were presented, in this meeting, which will aid the understanding of autophagy regulation at the molecular level
- Format: Kindle Edition
- File Size: 956 KB
- Print Length: 34 pages
- Publisher: Honnao (January 2, 2014)
- Sold by: Amazon Digital Services, Inc.
- Language: English
- ASIN: B00HNR1M8M
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Contents
Abstract
FLOW CYTOMETRIC MEASUREMENT OF CELL ORGANELLE PHAGY
AUTOPHAGY AND NEURODEGENERATION
A ROLE FOR Rab8 AND AUTOPHAGY IN THE REGULATION OF SYNAPSE GROWTH
AUTOPHAGY AS A BARRIER TO VIRAL AND NON-VIRAL GENE DELIVERY
17-BETA-ESTRADIOL AND PROGESTERONE ENHANCE EXPRESSION OF AUTOPHAGIC GENES IN BOVINE MAMMARY EPITHELIAL CELLS
INDUCTION OF AUTOPHAGY IN CHRONIC MYELOID LEUKAEMIA FOLLOWING TREATMENT WITH TYROSINE KINASE INHIBITORS MAY CONTRIBUTE TO DISEASE PERSISTENCE
THE CORE AUTOPHAGY PROTEIN ATG4B IS CRITICAL TO THE SURVIVAL OF LEUKEMIC STEM/PROGENITOR CELLS AND PREDICTS CLINICAL OUTCOMES OF CML PATIENTS TREATED WITH IMATINIB THERAPY
DYNAMIC ASSOCIATION OF THE ULK1 COMPLEX WITH OMEGASOMES DURING AUTOPHAGY INDUCTION
MOLECULAR MECHANISMS OF MAMMALIAN AUTOPHAGY
AUTOPHAGY IN HOST-PATHOGEN INTERACTION
Executive summary
About the Author
About the Chair
About the Speakers
David Rubinsztein
Agnes Foeglein
Sean T Sweeney
Sharon A. Tooze
Tom Wileman
Abstracts of oral and poster presentations
17BETA-ESTRADIOL AND PROGESTERONE ENHANCE EXPRESSION OF AUTOPHAGIC GENES IN BOVINE MAMMARY EPITHELIAL CELLS.
INDUCTION OF AUTOPHAGY IN CHRONIC MYELOID LEUKAEMIA FOLLOWING TREATMENT WITH TYROSINE KINASE INHIBITORS MAY CONTRIBUTE TO DISEASE PERSISTENCE
THE CORE AUTOPHAGY PROTEIN ATG4B IS CRITICAL TO THE SURVIVAL OF LEUKEMIC STEM/PROGENITOR CELLS AND PREDICTS CLINICAL OUTCOMES OF CML PATIENTS TREATED WITH IMATINIB THERAPY
DYNAMIC ASSOCIATION OF THE ULK1 COMPLEX WITH OMEGASOMES DURING AUTOPHAGY INDUCTION
AGGREGATION OF MISFOLDED PROTEINS AND THEIR CLEARANCE BY AUTOPHAGY; RELEVANCE TO NEUROPSYCHIATRIC DISEASES
17-BETA-ESTRADIOL AND PROGESTERONE REGULATES AUTOPHAGY DURING FUNCTIONAL DEVELOPMENT OF ALVEOLAR STRUCTURES FORMED BY BOVINE MAMMARY EPITHELIAL CELLS CULTURED IN 3D SYSTEM
CLEARANCE OF DYING RETINAL PIGMENT EPITHELIAL (RPE) CELLS BY PROFESSIONAL AND NON-PROFESSIONAL PHAGOCYTES AS IN VITRO MODEL FOR AGE-RELATED MACULAR DEGENERATION (AMD)
STIMULATION OF AUTOPHAGY AS A POTENTIAL TREATMENT FOR CHONDRODYSPLASIA CAUSED BY COLLAGEN X MUTATIONS
CROSS-TALK BETWEEN AUTOPHAGY AND APOPTOSIS IN LONG-TERM CULTURES OF BLOOD NEUTROPHILS
EXAMINATION OF COMMON GENE TARGETS FOR β-CATENIN AND NFkB PROTEINS IN IMATINIB SENSITIVE AND RESISTANT K562 CELL LINES
EFFECT OF NILOTINIB ON INFLAMMATORY MARKERS IN LPS/IFNγ ACTIVATED RAW 264.7 MACROPHAGES
INTERACTION OF NFΚB AND AKIRIN PROTEINS IN IMATINIB RESISTANT AND SENSITIVE K562 CELL LINES.
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